ScanFlo

Resources/Protocols

Orbits

Imaging the optic nerve and globe in a bed of bright orbital fat. Fat suppression is the whole battle — without it the nerve and any inflammation disappear — and the sinuses next door make that suppression fail unless you plan for it.

Protocols/Brain

When this study is used

  • Optic neuritis or unexplained visual loss.
  • Orbital mass, proptosis or thyroid eye disease.
  • Suspected optic-nerve glioma or meningioma.

Patient & coil positioning

  • Head-first supine in a head coil, eyes closed and still; instruct the patient to fix gaze straight ahead to reduce eye motion.
  • Centre on the glabella; immobilise firmly.
  • High-channel coil for the resolution small structures need.

Localizer

Three-plane localizer. The dedicated sequences use thin slices and a small FOV over the orbits.

Sequence planning

Each sequence below lists how the slice group is positioned — the reference line it follows, its coverage, and why it earns its place in the protocol. Plan against the localizer, not by eye.

Axial T2 fat-sat

Axial

Planning — Parallel to the optic nerve (slightly oblique to the hard palate) on the sagittal, so the tortuous nerve is 'unravelled' along its length. Both orbits.

Why — Optic-nerve signal and orbital contents against suppressed fat.

Coronal STIR

Coronal

Planning — Perpendicular to the optic nerve.

Why — Uniform fat suppression immune to sinus field-inhomogeneity — the reliable oedema detector here.

Axial/coronal/sagittal T1 + contrast fat-sat

Multi-plane

Planning — Matched thin slices post-gadolinium.

Why — Enhancing optic neuritis or an orbital lesion.

Representative parameters

Ranges, not commandments — field strength, coil and vendor move the numbers. What must not move is understanding why each sits where it does.

SequenceSliceNotes
T2 fat-sat axial≤3 mmAlong the optic-nerve axis
STIR coronal≤3 mmRobust to sinus inhomogeneity
T1 +C fat-sat≤3 mmEnhancement, three planes

Artifacts & how to fix them

ArtifactTypical causeMitigation
Black holes (susceptibility)Air–tissue interface at the sinusesUse STIR (frequency-independent); smaller voxels
Failed spectral fat-satOff-resonance near the orbitsSTIR instead of spectral fat-sat
Eye-motion blurRoving gazeFix gaze straight ahead; eyes closed and still

What am I looking at?

You are not reporting the study, but recognising pathology helps you keep it in the field of view and know when to add a sequence or contrast. How the common findings read on the console:

FindingSequence to checkHow it reads
Optic neuritisCoronal STIR / T2 FSSwollen, bright optic nerve against dark suppressed fat
Optic-nerve glioma / meningiomaT1 +C fat-satEnhancing, thickened nerve or sheath
Thyroid eye diseaseCoronalEnlarged extraocular muscle bellies sparing the tendons

Review checklist

  • Axials 'unravel' the optic nerve along its length.
  • STIR used where spectral fat-sat would fail near the sinuses.
  • Post-contrast in three planes for a suspected lesion.
  • Eye motion controlled with a fixed forward gaze.

Mistakes that cost repeats

  • Spectral fat-sat failing near the sinuses and hiding the nerve.
  • Nerve not profiled along its axis, so its length isn't shown.
  • Eye motion blurring the thin slices.
  • Susceptibility black-holes over pathology on gradient-echo sequences.

Common questions

Why is fat suppression critical in orbit imaging?
Orbital fat is bright on both T1 and T2. Without suppression it drowns out the optic nerve and any inflammation, so fat-suppressed sequences — often STIR — are essential to see the nerve at all.
Why prefer STIR over spectral fat-sat in the orbits?
The air-filled sinuses beside the orbit distort the local magnetic field, which makes frequency-based spectral fat-sat fail patchily. STIR suppresses fat by inversion time rather than frequency, so it stays uniform in that 'messy' environment.
How are the axial slices angled for the optic nerve?
Parallel to the optic nerve — slightly oblique to the hard palate — so the naturally tortuous nerve is straightened out and its full length is shown in fewer slices.

Try it in ScanFlo: Run a spectral fat-sat T2 and a STIR through the orbits and compare the suppression near the sinuses. STIR stays uniform where the spectral sequence breaks up — that is why it is the orbit workhorse.

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